[1] Meng XM,Chung AC,Lan HY.Role of the TGF-β/BMP-7/Smad pathways in renal diseases[J].Clin Sci(Lond),2013,124(4):243-254.
[2] Das R,Xu S,Quan X,et al.Upregulation of mitochondrial Nox4 mediates TGF-β-induced apoptosis in cultured mouse podocytes[J].Am J Physiol Renal Physiol,2014,306(2):155-167.
[3] Wang Z,Han Z,Tao J,et al.Transforming growth factor-β1 induces endothelial-to-mesenchymal transition via Akt signaling pathway in renal transplant recipients with chronic allograft dysfunction[J].Ann Transplant,2016,21:775-783.
[4] Li ZL,Shi Y,Ding Y,et al.Dietary oxidized tyrosine (O-Tyr) stimulates TGF-β1-induced extracellular matrix production via the JNK/p38 signaling pathway in rat kidneys[J].Amino Acids,2017,49(2):241-260.
[5] Loeffler I, Wolf G.Transforming growth factor-β and the progression of renal disease[J].Nephrol Dial Transplant,2014,29(Suppl 1):i37-i45.
[6] Braun F,Becker JU,Brinkkoetter PT.Live or let die:Is there any cell death in podocytes[J].Semin Nephrol,2016,36(3):208-219.
[7] Kriz W,Lemley KV.A potential role for mechanical forces in the detachment of podocytes and the progression of CKD[J].J Am Soc Nephrol,2015,26(2):258-269.
[8] Kriz W,Shirato I,Nagata M,et al.The podocyte's response to stress: the enigma of foot process effacement[J].Am J Physiol Renal Physiol,2013,304(4):F333-F347
[9] Dong C,Zheng H,Huang S,et al.Heme oxygenase-1 enhances autophagy in podocytes as a protective mechanism against high glucose-induced apoptosis[J].Exp Cell Res,2015,337(2):146-159.
[10] Tharaux PL,Huber TB.How many ways can a podocyte die[J].Semin Nephrol,2012,32(4):394-404.
[11] Eid AA.Mammalian target of rapamycin regulates Nox4-mediated podocyte depletion in diabetic renal injury[J].Diabetes,2013,62(8):2935-2947.
[12] Huang H,You Y,Lin X,et al.Inhibition of TRPC6 signal pathway alleviates podocyte injury induced by TGF-β1[J].Cell Physiol Biochem,2017,41(1):163-172.
[13] Ka SM,Yeh YC,Huang XR,et al.Kidney-targeting Smad7 gene transfer inhibits renal TGF-β/MAD homologue (SMAD) and nuclear factor κB (NF-κB) signalling pathways,and improves diabetic nephropathy in mice[J].Diabetologia,2012,55(2):509-519.
[14] Xu L,Zhang P,Guan H,et al.Vitamin D and its receptor regulate lipopolysaccharide-induced transforming growth factor-β,angiotensinogen expression and podocytes apoptosis through the nuclear factor-κB pathway[J].J Diabetes Investig,2016,7(5):680-688.
[15] Chen J,Chen JK,Harris RC.EGF receptor deletion in podocytes attenuates diabetic nephropathy[J].J Am Soc Nephrol,2015,26(5):1115-1125.
[16] Chen Z,Wan X,Hou Q,et al.GADD45B mediates podocyte injury in zebrafish by activating the ROS-GADD45B-p38 pathway[J].Cell Death Dis,2016,7(1):e2068.
[17] Liu L,Lin W,Zhang Q,et al.TGF-β induces miR-30d down-regulation and podocyte injury through Smad2/3 and HDAC3-associated transcriptional repression[J].J Mol Med(Berl),2016,94(3):291-300.