[1] Jankovic J, Tan EK. Parkinson's disease: etiopathogenesis and treatment[J]. J Neurol Neurosurg Psychiatry, 2020, 91(8): 795-808.
[2] Badanjak K, Fixemer S, Smajic S, et al. The Contribution of Microglia to Neuroinflammation in Parkinson's Disease[J]. Int J Mol Sci, 2021, 22(9): 4676.
[3] Li W, He PC, Huang YG, et al. Selective autophagy of intracellular organelles: recent research Advances[J]. Theranostics, 2021, 11(1): 222-256.
[4] 和璐璐, 樊慧杰, 李艳荣, 等. 线粒体功能障碍与帕金森病[J]. 中国实用神经疾病杂志, 2023, 26(10): 1305-1310.
[5] Borsche M, Pereira SL, Klein C, et al. Mitochondria and Parkinson's disease: clinical, molecular, and translational aspects[J]. J Parkinsons Dis, 2021, 11(1): 45-60.
[6] Liu K, Zhao Q, Sun HY, et al. BNIP3 (BCL2 interacting protein 3) regulates pluripotency by modulating mitochondrial homeostasis via mitophagy[J]. Cell Death Dis, 2022, 13(4): 334.
[7] 陈先文, 王刚, 陈生弟. 帕金森病药物治疗研究进展[J]. 重庆医科大学学报, 2024, 49(5): 542-547.
[8] Yu W, Ilyas I, Aktar N, et al. A review on therapeutical potential of paeonol in atherosclerosis[J]. Front Pharmacol, 2022, 13: 950337.
[9] 朱彤, 曾广红, 周杰. 丹酚酸B在帕金森病动物模型中的神经保护作用及临床应用进展[J]. 长春中医药大学学报, 2024, 40(3): 350-354.
[10] Angelopoulou E, Paudel YN, Papageorgiou SG, et al. Environmental Impact on the Epigenetic Mechanisms Underlying Parkinson's Disease Pathogenesis: A Narrative Review[J]. Brain Sci, 2022, 12(2): 175.
[11] 胡志雷. Optineurin通过激活线粒体自噬减轻氧化应激所致椎间盘退变的作用机制研究[D]. 重庆: 中国人民解放军陆军军医大学, 2022.
[12] Li D, Huang LT, Zhang CP, et al. Insights Into the Role of Platelet-Derived Growth Factors: Implications for Parkinson's Disease Pathogenesis and Treatment[J]. Front Aging Neurosci, 2022, 14: 890509.
[13] Razali K, Kumar J, Mohamed WMY. Characterizing the adult zebrafish model of Parkinson's disease: a systematic review of dynamic changes in behavior and physiology post-MPTP administration[J]. Front Neurosci, 2024, 18: 1432102.
[14] Yao SQ, Xu ZP, Guo WX. Neuroprotective effect of Cistanche deserticola glycosides in MPTP-Induced Parkinson's disease mouse model involves Nrf2 activation[J].J Toxicol Environ Health A, 2025 , 8: 1-13.
[15] Mustapha M, Taib CNM. MPTP-induced mouse model of Parkinson's disease: A promising direction of therapeutic strategies[J]. Bosn J Basic Med Sci, 2021, 21(4): 422-433.
[16] 郑小惠, 刘坤, 辛航阔, 等. 线粒体自噬在神经退行性疾病中调控的研究进展[J]. 中国畜牧兽医, 2023, 50(2): 490-499.
[17] Panicker N, Ge P, Dawson VL, et al. The cell biology of Parkinson's disease[J]. J Cell Biol, 2021, 220(4): e202012095.
[18] Chavda V, Lu B. Reverse Electron Transport at Mitochondrial Complex I in Ischemic Stroke, Aging, and Age-Related Diseases[J]. Antioxidants (Basel), 2023, 12(4): 895.
[19] Teleanu DM, Niculescu AG, Lungu II, et al. An Overview of Oxidative Stress, Neuroinflammation, and Neurodegenerative Diseases[J]. Int J Mol Sci, 2022, 23(11): 5938.
[20] Jin W, Kam KM, Lee SW, et al. Peroxiredoxin 2 Ameliorates AβO-Mediated Autophagy by Inhibiting ROS via the ROS-NRF2-p62 Pathway in N2a-APP Swedish Cells[J]. Antioxidants (Basel), 2022, 11(10): 1889.
[21] Boyd RJ, Avramopoulos D, Jantzie LL, et al. Neuroinflammation represents a common theme amongst genetic and environmental risk factors for Alzheimer and Parkinson diseases[J]. J Neuroinflammation, 2022, 19(1): 223.
[22] Heidari A, Yazdanpanah N, Rezaei N. The role of Toll-like receptors and neuroinflammation in Parkinson's disease[J]. J Neuroinflammation, 2022, 19(1): 135.
[23] Simon DK, Tanner CM, Brundin P. Parkinson Disease Epidemiology, Pathology, Genetics, and Pathophysiology[J]. Clin Geriatr Med, 2020, 36(1): 1-12.
[24] Choubey V, Zeb A, Kaasik A. Molecular Mechanisms and Regulation of Mammalian Mitophagy[J]. Cells, 2021, 11(1): 38.
[25] Cunnane SC, Trushina E, Morland C, et al. Brain energy rescue: an emerging therapeutic concept for neurodegenerative disorders of ageing[J]. Nat Rev Drug Discov, 2020, 19(9): 609-633.
[26] 陈思, 陆斌, 毕堃, 等. 甘草酸通过激活TRPV4诱导线粒体自噬抑制脑胶质瘤细胞的生长[J]. 现代肿瘤医学, 2023, 31(15): 2776-2781.
[27] Iorio R, Celenza G, Petricca S . Mitophagy: Molecular Mechanisms, New Concepts on Parkin Activation and the Emerging Role of AMPK/ULK1 Axis[J]. Cells, 2021, 11(1): 30.
[28] Khan T, Waseem R, Zehra Z, et al. Mitochondrial Dysfunction: Pathophysiology and Mitochondria-Targeted Drug Delivery Approaches[J]. Pharmaceutics, 2022, 14(12): 2657.
[29] Ying YC, Jiang PF . Research progress on transient receptor potential melastatin 2 channel in nervous system diseases[J]. Zhejiang Da Xue Xue Bao Yi Xue Ban, 2021, 50(2): 267-276.
[30] Zhang LJ, Dai L, Li DY. Mitophagy in neurological disorders[J]. J Neuroinflammation, 2021, 18(1): 297.
[31] Liu L, Li Y, Chen G, et al. Crosstalk between mitochondrial biogenesis and mitophagy to maintain mitochondrial homeostasis[J]. J Biomed Sci, 2023, 30(1): 86.
[32] Li EM, Li XY, Huang J, et al. BMAL1 regulates mitochondrial fission and mitophagy through mitochondrial protein BNIP3 and is critical in the development of dilated cardiomyopathy[J]. P C, 2020, 11(9): 661-679.
[33] Madhu V, Hernandez-Meadows M, Boneski PK, et al. The mitophagy receptor BNIP3 is critical for the regulation of metabolic homeostasis and mitochondrial function in the nucleus pulposus cells of the intervertebral disc[J]. Autophagy, 2023, 19(6): 1821-1843.
[34] Guo ZL, Yang Y, Li L, et al. The novel prolyl hydroxylase-2 inhibitor caffeic acid upregulates hypoxia inducible factor and protects against hypoxia[J]. Eur J Pharmacol, 2022, 934: 175307.
[35] Wang DX, Yang Y, Huang XS, et al. Pramipexole attenuates neuronal injury in Parkinson's disease by targeting miR-96 to activate BNIP3-mediated mitophagy[J]. Neurochem Int, 2021, 146: 104972.
[36] 张超颖, 苗纪飞, 刘霞, 等. 丹皮酚对脂多糖刺激的RAW264.7细胞炎症与自噬的影响[J]. 中国中医基础医学杂志, 2020, 26(6): 768-772, 821.
[37] 孙胜男, 和璐璐, 秦劭晨, 等. 丹皮酚对帕金森病模型中人神经母细胞瘤细胞的保护作用及其初步机制[J]. 解放军医学杂志, 1-10[2025-01-17].
[38] 刘超. 丹皮酚通过Pink1/Parkin通路调控细胞自噬抑制心梗后心室重构[D]. 承德: 承德医学院附属医院, 2021.
[39] 刘少静, 沈晶晶, 卢颖, 等. 丹皮酚、绿原酸和没食子酸复配物的体外抗氧化活性[J]. 化工科技, 2022, 30(1): 5-8.
[40] Zhang YC, Wu XY, Wang XH, et al. Grey Relational Analysis Combined With Network Pharmacology to Identify Antioxidant Components and Uncover Its Mechanism From Moutan Cortex[J]. Front Pharmacol, 2021, 12: 748501.
[41] Niu YT, Zhang J, Shi DH, et al. Glycosides as Potential Medicinal Components for Ulcerative Colitis: A Review[J]. Molecules, 2023, 28(13): 5210.
[42] Liang F, Tian XY, Ding LN. Daphnetin modulates GLP-1R to alleviate cognitive dysfunction in diabetes: implications for inflammation and oxidative stress[J]. Front Pharmacol, 2024, 15: 1438926.
[43] 杜松, 范佩娟, 王夏, 等. 丹皮酚在治疗帕金森病的药理作用机制研究进展[J]. 辽宁中医药大学学报, 1-11[2025-01-17].
[44] 何玉珏. 丹皮酚及其两种同分异构体与DPPC脂质体相互作用研究[D]. 北京: 北京中医药大学, 2021.
[45] Jin HY, Wang MY, Wang JM, et al. Paeonol attenuates isoflurane anesthesia-induced hippocampal neurotoxicity via modulation of JNK/ERK/P38MAPK pathway and regulates histone acetylation in neonatal rat[J]. J Matern Fetal Neonatal Med, 2020, 33(1): 81-91.
[46] 李港澳, 李侃, 陈超, 等. 脑血疏口服液通过p38MAPK通路抑制帕金森病大鼠黑质神经炎症反应[J]. 中国老年学杂志, 2019, 39(7): 1662-1666.
[47] Ramazi S, Fahanik-Babaei J, Mohamadi-Zarch SM, et al. Paeonol exerts neuroprotective and anticonvulsant effects in intrahippocampal kainate model of temporal lobe epilepsy[J]. J Chem Neuroanat, 2022, 124: 102121.
[48] 王训翠, 朱国旗, 赖桂华, 等. 丹皮酚对MPP+诱导的SH-SY5Y细胞线粒体自噬和死亡的保护作用[J]. 中国药理学通报, 2018, 34(12): 1655-1661.
[49] 周华勇, 玉萍, 杨旭, 等. 丹皮酚通过调节p38丝裂原活化蛋白激酶对缺血性脑卒中大鼠神经炎症的影响[J]. 心脑血管病防治, 2024, 24(2): 16-20, 65.